Izimpawu zengqondo ku-Polycystic Ovarian Syndrome (PCOS) yinkinga yemizwa.
Isikhathi sokufunda esilinganiselwe: 14 imizuzu
Izinkinga ngenkumbulo, ukugxila, nokufunda ezibonwa kwabesifazane abane-PCOS yizinkinga zemizwa. Futhi yingakho ukuqhubeka namaphilisi okulawula inzalo kungeke kulungise.
Ngizama ukuhlala esihlokweni kule bhulogi. Ngifuna ngempela i-Google ithole umbono ocace kakhulu mayelana nokuthi le bhulogi imayelana nani ukuze isize abantu bayithole futhi ivumele abantu bazi zonke izindlela abangazizwa bengcono ngazo. Ngakho ngaba manqikanqika ukubhala isihloko mayelana ne-PCOS, ngokwesaba ukuthi i-algorithm ngeke iqonde ukuthi izokubonani njengoshintsho esihlokweni.
Kodwa ake ngibhale lokhu ngokucace kakhulu ku-algorithm ye-Google kanye nawe, mhlawumbe umuntu one-PCOS obhekene nenkungu yobuchopho.
Izimpawu zokucabanga ohlupheka ngazo ngenxa ye-PCOS yakho, oyishoyo futhi oyikhomba njengenkungu yobuchopho, azikho mayelana nesimo sakho se-hormone futhi ngeke usule inkungu yobuchopho bakho noma i-PCOS yakho usebenzisa amaphilisi okulawula ukuzalwa. Ngidinga wena, kanye ne-Google, ukuthi niqonde ukuthi inkungu yobuchopho oyibonayo ingenxa yokuntuleka kwamandla okwenzeka engqondweni yakho.
Futhi kungenzeka usemncane kakhulu uma une-PCOS. Akekho umuntu oseminyakeni yawo-20 no-30 okufanele abhekane nezimpawu zenkungu yobuchopho.
Ake sixoxe ngokuthi kungani inkungu yobuchopho iza ne-PCOS
I-PCOS kanye nokumelana ne-insulin
Uma wenza noma yiluphi ucwaningo lwakho mayelana ne-PCOS yakho, khona-ke uyazi ukuthi ikhula esimweni sokumelana ne-insulin. Ukumelana ne-insulin yisimo esingadala izifo eziningi ezingelapheki eziningi kubantu abaningi nokuthi lezo zifo zibonakala kanjani kungenzeka zihlobene nezakhi zofuzo kanye/noma lapho kukhula ukumelana ne-insulin okuqondene nezicubu ezithile. Iqiniso elincane elaziwa ukuthi i-insulin iyihomoni eyinhloko enethonya kumahomoni ocansi futhi ikakhulukazi, ukuguqulwa kwamanye amahomoni abe amanye amahomoni. Yilokhu okungahambi kahle ku-PCOS.
Kulabo abasha ekuqondeni kwabo ukumelana ne-insulin, isisekelo esiyisisekelo siwukuthi ngenxa yempilo yonke yokudla okugaywe kakhulu, noma ngisho nokudla ama-carbohydrate amaningi kunalokho esingakwazi ukukuthwala ngenxa yanoma yisiphi isizathu, ingxenye ebalulekile yamangqamuzana ethu iyaphuka. Ama-insulin receptors. Kungumsebenzi we-insulin ukusunduza i-glucose ibe amaseli ukuze iguqulwe ibe uphethiloli. Kodwa uma kunenani elingapheli le-glucose egazini, eligcina i-insulin ivuliwe njalo futhi iphezulu, ama-receptors aphelelwa amandla. Iglucose ayikwazi ukuphushwa kumaseli ngendlela efanele bese isetshenziswa. Lokhu kushiya amazinga ayingozi futhi avuvukalayo eglucose esegazini elenga futhi enze umonakalo omkhulu ezicutshini njengoba umzimba uzama ukuwususa.
Ngidinga ukukutshela ukuthi umzimba wakho wenza kangakanani hhayi njengoshukela oweqile egazini lakho. Ifuna kuphela inani lethisipuni nganoma yisiphi isikhathi. Umzimba wakho uzogcina i-glucose ezicutshini ezithile njengemisipha, isibindi, kanye nengcosana ezinso. Kodwa uma ungeyena umuntu ovocavoca umzimba okhuthele oqeda lezi zitolo futhi ungakwazi ukucwilisa i-glucose eyengeziwe emsipha, ivele ilenge egazini lakho. Yebo, ingqondo yakho isebenzisa i-glucose kodwa ngenani elincane. Inani elincane kakhulu nganoma isiphi isikhathi esisodwa kunesiphuzo esinoshukela osanda kusiphuza, noma wonke lawo mashibhisi ommbila owawunawo enziwe iglucose ngokushesha ngemva kokudla. Cha, i-fiber ocabanga ukuthi yayikulawo mashibhisi ommbila ayilinciphisi izinga lokwenziwa kwayo ibe i-glucose kunoma yimuphi umphumela oncomekayo. Ishaye igazi lakho njengebhomu.
Uma ungumfundi onolwazi, kungenzeka ukuthi uthi ima kancane! Ukuhanjiswa kweglucose ebuchosheni kuncike kakhulu ku-insulin. Ngingakuthola kanjani ukumelana ne-insulin ebuchosheni, impela?
Ukuhlobana okuqondile phakathi kokumelana ne-insulin kanye ne-glycose hypometabolism ebuchosheni akucaci, kodwa mfana oh boy kunendaba yini nomsebenzi we-synaptic, i-metabolism yobuchopho, namazinga e-neuroinflammation. Konke okungenzeka kube nomthelela esimweni se-hypometabolism okubangela izimpawu zokuqaphela kwabesifazane abane-PCOS.
Ake ngikubonise.
Uyazibona zonke izingxenye esithombeni esingenhla ezifundeka ngokuthi “IR”? I-IR, kulesi sithombe, ibhekise ebukhoneni be-insulin receptors. Ukusebenza kwazo zonke lezi zingxenye kungaba ukumelana ne-insulin futhi ngenxa yalokho, kungakwazi ukuthola amandla kushukela. Lena yonke imisebenzi ebaluleke kakhulu yezesekeli empilweni yobuchopho, ukugcinwa nokusebenza.
Ngingabhala indatshana yebhulogi kalula ngokubaluleka kwazo zonke izakhiwo nemisebenzi eshiwo kulesi sithombe. Udinga lezi ukuze zisebenze kahle ukuze usekele ukusebenza kobuchopho bakho. Futhi ngifuna ukwazi lokhu ukuze uma ufunda okuthile ku-inthanethi okukhuluma ngokuthi ubuchopho bungayisebenzisi kanjani i-insulin ukuthatha amandla, uqonde ukuthi leso sitatimende sinombono omfishane ngokudabukisayo mayelana nesidingo sokusebenza kokwamukela i-insulin ngaphakathi. umgoqo wegazi-wobuchopho kanye namaseli e-neuronal ebuchosheni.
Ungabona kulesi sithombe esihle kakhulu, ukuthi uma lezi zakhiwo ze-neuronal ezithembele kubathuthi be-insulin enempilo zingawatholi amandla, izakhiwo ze-neuronal ezenza ingqamuzana lisebenze zizohluleka ukuthatha amandla ngendlela enempilo futhi eqhubekayo. Umugqa ongezansi.
I-Brain Hypometabolism - kungani unenkungu yobuchopho
Ngakho-ke nansi indlela izimpawu zokuqonda ohlangabezana nazo ku-PCOS zingokwemizwa hhayi i-hormonal ngokwesho. Ubuchopho bakho buqala ukumelana ne-insulin, futhi usuqala ukungakwazi ukusebenzisa i-glucose njengomthombo wamafutha. Futhi yingakho unenkungu yobuchopho, izinkinga zokukhumbula, futhi mhlawumbe nezinkinga zemizwa ezingewona umthelela oqondile wokuphazamiseka kwehomoni yakho.
Ubuchopho obugcwele i-glucose engakwazi ukuyiphusha emangqamuzaneni ubuchopho obuvutha umlilo obunokuvuvukala kwe-neuroinflammation. I-Neuroinflammation idala ukungalingani kwe-neurotransmitter, isebenzisa ama-micronutrients abalulekile njalo uzama ukulungisa umonakalo walesi simo futhi isuse uchungechunge lwezimo ze-neurodegenerative ezizohlanganisa izimpawu zakho zokuqonda. Lokhu kuthinta isimo sakho sengqondo futhi kuqhubekisele phambili imizwa yokukhathazeka nokucindezeleka, ngaphandle kwe-testosterone yakho namanye amazinga ehomoni.
Uma ungathanda ukufunda ukuthi i-neuroinflammation inomthelela kanjani ekucindezelekeni, uzofuna ukufunda okuthunyelwe kwebhulogi okungezansi:
Konke kuxhumene kakhulu.
Kungani ngingatshelwanga lokhu?!
Angazi kungani singabatsheli abantu besifazane lokhu. Angazi ukuthi kungani singasebenzisi indlela yokudla nendlela yokuphila ukwelapha i-PCOS (kanye nenkungu yobuchopho eyibangelayo) njengembangela eyisisekelo yokumelana ne-insulin emithini evamile. Kodwa indaba ye-hypometabolism yobuchopho kwabesifazane abane-PCOS ibhalwe kahle.
Isilinganiso seminyaka yobudala lapho i-hypometabolism yobuchopho ibonwe kwabesifazane abane-PCOS isencane impela futhi inesilinganiso seminyaka yobudala engama-25 futhi ukuncipha kwekhono lobuchopho lokusebenzisa i-glucose kwehliswe phakathi kuka-9-14%.
Lokho akuzwakali njengokuningi. Kodwa iyinani elilimaza ingqondo. Ikakhulukazi uma ucabanga ukuthi kuze kufike ku-40% wawo wonke amandla umzimba wakho owenzayo asetshenziswa ebuchosheni. Ubuchopho bukhungathekile ngenxa yokuntuleka kwamandla.
Imiphumela yethu ibonisa ukuthi abesifazane abanesisindo esivamile esingalashwa abane-PCOS babene-glucose ephansi yobuchopho besifunda ngephethini efana naleyo ebonakala kubantu abadala futhi, kancane kancane, ekuqaleni kwe-AD [i-Alzheimer's Disease].
Castellano, CA, Baillargeon, JP, Nugent, S., Tremblay, S., Fortier, M., Imbeault, H., … & Cunnane, SC (2015). I-regional brain glucose hypometabolism kwabesifazane abasebasha abane-polycystic ovary syndrome: isixhumanisi esingaba khona sokumelana ne-insulin okuncane. PLoS One, 10(12), i-e0144116. https://doi.org/10.1371/journal.pone.0144116
Izikena zobuchopho ezikala ukumukeka kweglucose ukuze zibe uphethiloli ebuchosheni bathole lokho abesifazane abane-PCOS babenciphise umzimba wamandla obuchopho ku-frontal, parietal kanye ne-temporal cortex. Futhi nakuba laba besifazane banikezwa izivivinyo zokuqonda ezanquma umsebenzi wabo “njengovamile”, abesifazane abagcinanga nje ngokubonisa leli khono elinciphile lokuthatha uphethiloli kulezi zindawo zobuchopho besebenzisa amasu e-imaging kodwa futhi babekhononda ngokuthi inkumbulo yabo yokusebenza yayizizwa ikhubazekile.
Futhi lesi ngesinye sezizathu zokuthi kungani emsebenzini wami nabesifazane abanenkungu yobuchopho, sisebenzela ukubavumela ukuthi baqinisekise okuhlangenwe nakho kwabo. Uyawazi ubuchopho bakho.
Angikhathali uma ukuhlolwa kwengqondo kowesifazane kubuye ngokwejwayelekile, noma udokotela wabo uthe akukho okumele bakhathazeke ngakho. Akunandaba ukuthi udokotela wabo uthe "ukuguga okuvamile" (engingathemba ukuthi ngeke njengoba sikhuluma ngabesifazane abaneminyaka engu-20 ubudala abane-PCOS nezimpawu zenkungu yobuchopho!).
Abesifazane bayazazi. Uyazazi wena. Uyazi lapho ingqondo yakho izizwa kahle futhi uyazi lapho ingasebenzi kahle njengoba yayike yenza. Kungenzeka ukuthi ingqondo yakho ayikaze ibe mnandi futhi uyazi ukuthi kufanele isebenze kangcono. Lokho kuvumelekile. Uyakunaka futhi unelungelo lokwazi zonke izindlela ongayenza ukuthi uzizwe ungcono. Futhi yilokho okushiwo yile bhulogi.
Ngiyilungisa kanjani ingqondo yami lapho ngine-PCOS?
Kufanele siguqule uphethiloli wobuchopho bakho busuke ku-glucose iye kuma-ketone.
Ama-ketones ayakwazi ukuya ngqo kumangqamuzana obuchopho alambile futhi asetshenziswe njengomthombo ohlukile wamafutha ku-glucose. Kunezingxenye ezimbalwa zobuchopho bakho eziyohlale zidinga ukufinyelela ku-glucose. Kodwa awudingi ukudla i-glucose ukuze uphese lezo zingxenye ezincane zobuchopho bakho ezisebenzisa i-glucose. Isibindi sakho siyakwazi ukwenza yonke i-glucose oyidingayo ukuze ifuthe lezo zingxenye zobuchopho ngomshini obizwa ngokuthi i-gluconeogenesis.
Lapho unikeza ubuchopho ama-ketones ukuthi ashise ngokuqondile ukuze uthole uphethiloli, izindawo ze-glucose hypometabolism ziyavuka futhi ziqale ukusebenza futhi. Ngokushesha ama-neuron asebuchosheni bakho angakwazi ukwenza amabhethri eseli engeziwe (mitochondria) futhi angasebenzisa wonke lawo mandla amangalisayo ukucabanga, ukukhumbula, ukugxila kanye nokuzwa. Ama-neurons azoba namandla okulungisa umonakalo we-neuronal.
Futhi zonke izingxenye zamangqamuzana amancane nemisebenzi oyibone esithombeni esimangalisayo esingenhla yenzeka ngokuthanda ama-ketone ukuze abe namafutha. Ziyakwazi ukukhulisa ukusebenza kwalezo zakhiwo noma zivele zifakwe futhi zisetshenziselwe uphethiloli kalula, zidlule lawo ma-insulin receptors aphukile.
Kunezindlela ezimbili zokuthola ama-ketone ukuze uhlenge umsebenzi wakho wokuqonda.
- Yidla izinto ezinikeza uphethiloli we-ketone (isb, uwoyela we-MCT kanye/noma usawoti we-ketone)
- Khawulela ukusetshenziswa kwama-carbohydrate ngokwanele ukuthi amazinga akho e-insulin ehle ngokwanele ukuthi ungenza ama-ketone ngamafutha okudla noma izitolo zamafutha zomzimba wakho.
Nakhu okudingeka ukuqonde uma une-PCOS futhi ufuna ukwelapha zonke izimpawu, hhayi nje inkungu yobuchopho bakho. Ngiyazi uma une-PCOS ukuthi unezinye izimpawu ezinzima kakhulu ongaphila nazo ngaphandle kwenkungu yobuchopho. Futhi ukwelapha lezo zimpawu, kuzodingeka ukhethe indlela yokwelapha yokudla. Ngoba imbangela eyinhloko ukuphikiswa kwe-insulin futhi ukuze uphulukise futhi uzizwe ukhahlela futhi uyamangalisa (okufanele!) uzodinga ukuphulukisa ukumelana kwakho ne-insulin. Futhi okuwukuphela kwendlela yokwenza lokho, mngane wami, ukukhawulela ukudla kwakho okunama-carbohydrate.
Kodwa nginenkinga yokudla! Angikwazi ukukhawulela!
Uma kutholwe ukuthi une-anorexia futhi ungabuyiselwa isisindo, lokho kungase kube njalo. Sisafunda ngocwaningo ukuthi ukukhawulelwa kwe-carbohydrate yokwelapha kungaba usizo yini kulesi sifo.
Kodwa nansi into. Abanye benu (ngokuqinisekile akubona bonke) abane-PCOS bahlushwa isisindo somzimba noma bangase babe nokukhuluphala ngokweqile. Ungase ube sekwelashweni kwengqondo ngenxa yengxenye yokuphazamiseka kwemizwelo yesifo sakho. Ungase ube nomelaphi okutshela ukuthi kuyingozi kuwe ukukhawulela noma yini. Ukuthi udinga ukugxila ekuziphatheni komzimba futhi usebenzise ukudla okunembile, futhi kungenzeka ukuthi bakuthole unesifo sokuzitika noma i-bulimia. Futhi ngakho-ke ukholelwa ukuthi ukudla okukhiqiza ama-ketone usebenzisa ukuvinjelwa kwe-carbohydrate yokwelapha kungase kube yingozi kuwe noma ngaphandle kwetafula ngoba kuzodingeka ukhawule noma unciphise i-macronutrient engabalulekile yama-carbohydrate.
Kodwa ngikutshela ukuthi udinga ukuthola umbono wesibili.
Izimpawu zokungadli kahle zivame ukwenzeka ngenxa yokumelana ne-insulin ngenxa yezinye izici zamahomoni (isb., Ukumelana ne-Leptin). Futhi unelungelo lokwazi ukuthi iziyaluyalu zokudla ngokuzitika futhi ngisho ne-bulimia zilashwa ngempumelelo ochwepheshe bezinkinga zokudla abaqeqeshwe kahle emhlabeni wonke besebenzisa izidlo ze-ketogenic. Kuthatha nje abelaphi abaningi kanye nezazi zokusebenza kwengqondo isikhathi eside ngempela ukuqonda nokushadisa isayense yengqondo kanye ne-biochemistry yokudla okunempilo ibe ukwelashwa okucabangela kokubili.
Ngakho-ke ungachofozi leli khasi ngaphambi kokwenza ucwaningo lwakho. Thola udokotela onolwazi oluncane, udokotela wengqondo, noma umelaphi kwenye yamasayithi ekhasini lezinsiza.
Uma ubuchopho bakho buthola ukunakekelwa kwalabo amnandi, abahlenga ukuqonda, i-neurotransmitter-bancing, i-inflammation-busting ketones, uzongibonga kakhulu.
Uma ufuna nje ukuzama uwoyela we-MCT kanye/noma usawoti we-ketone ukuze uzame ukuqondisa umzimba wakho wamandla obuchopho, ungakuzama nakanjani lokho. Kodwa ngifuna uqonde ukuthi lokhu ngeke kumise ezinye izimpawu zakho ezinzima, ezihlanganisa okulandelayo:
Lezo zimpawu zimayelana nokumelana ne-insulin kwezinye izicubu kanye nomthelela wamazinga aphezulu e-insulin anawo ekuphazamisekeni kwamahomoni akho ocansi.
Futhi ukuze labo babe ngcono, kufanele wehlise amazinga e-insulin aphezulu njalo. Ama-ketones angaphandle (isb, uwoyela we-MCT kanye/noma nosawoti weKetone) ngeke alinganise amahomoni akho. Ngeke banciphise noma benze amathegi esikhumba anyamalale. Ngeke bakwazi ukwenza umsebenzi odingekayo ukuze wenze izikhathi zakho zibe buhlungu futhi zibe buhlungu. Kukhona ukwelashwa kwe-PCOS, futhi kuwukudla kwe-ketogenic.
Ngakho-ke yebo, zama amanye ama-ketones angaphandle futhi ubone ukuthi ubuchopho bakho buzizwa bungcono yini. Kokuhlangenwe nakho kwami komtholampilo, lolu uhlobo lokungenelela olushaywe futhi oluphuthelwe ngaphandle koshintsho lwendlela yokudla. Abanye abantu bazizwa benamandla obuchopho, kanti abanye abazizwa lutho. Kunzima ukuthola umthamo oqondile ngezinye izikhathi. Futhi ngokuhlangenwe nakho kwami, ama-ketones angaphandle awasebenzi kahle endaweni evuvukala kakhulu njengaleyo edalwa ngokukhetha kokudla kanye nendlela yokuphila.
Sicela nje ungazami ama-ketones angaphandle bese unquma ukuthi ukudla kwe-ketogenic akuyona impendulo. Amafutha e-MCT nama-Ketone Salts awafani nokudla kwe-ketogenic. Kunemiphumela esebenzisa ukuvinjelwa kwe-carbohydrate yokwelapha (izidlo ze-ketogenic) ongeke ukuthole nge-exogenous ketone supplementation kuphela. Futhi uyazikweleta ukuzwa ubuchopho obunamandla aphakeme, ukuvuvukala okuncishisiwe, nama-hormone abhalansi kangcono nama-neurotransmitters. Wonke umuntu ufanelwe ukwazi ukuthi kunjani ukuba nobuchopho obusebenza ngokugcwele ngokusebenzisa izinqubo zokudla okunomsoco kanye nokusebenza kwengqondo okubandakanya ukudla okune-ketogenic okwakhiwa kahle kanye nezici zokwengezwa ezilungiselelwe wena kanye nezici zokuphila.
Ufanelwe ukuzizwa ungcono kakhulu kunawe.
Futhi uma umqondo wokudla okune-ketogenic uzwakala unzima kakhulu, yazi ukuthi nginohlelo lokukusiza kukho konke ukwehla nokwenyuka nokucatshangelwa koshintsho olunjalo lwendlela yokuphila ukuze uphathe inkungu yobuchopho. Ngisiza abesifazane ukuthi bahlehlise inkungu yobuchopho ngaso sonke isikhathi, kungakhathaliseki ukuthi yisiphi isizathu noma ukuxilongwa abatshelwe ukuthi kuyimbangela.
Uma ufuna ukufunda okwengeziwe ngohlelo lwami lwe-inthanethi ungafunda kabanzi lapha:
Takula umsebenzi wakho wokuqonda manje. Udinga ubuchopho obusebenza kahle ukuze uphile impilo yakho ngokugcwele futhi ube khona ngokomzwelo kanye nobuwena obuhle kakhulu ebudlelwaneni bakho obubalulekile.
Ngiyakuthembisa kuyenzeka.
Okubhekwayo
Arnold, SE, Arvanitakis, Z., Macauley-Rambach, SL, Koenig, AM, Wang, H.-Y., Ahima, RS, Craft, S., Gandy, S., Buettner, C., Stoeckel, LE, Holtzman, DM, & Nathan, DM (2018). Ukuphikiswa kwe-insulin yobuchopho ohlotsheni lwe-2 yesifo sikashukela kanye nesifo se-Alzheimer: Imiqondo kanye ne-conundrums. Ukubuyekezwa Kwemvelo. I-Neurology, 14(3), i-168-181. https://doi.org/10.1038/nrneurol.2017.185
Castellano, C.-A., Baillargeon, J.-P., Nugent, S., Tremblay, S., Fortier, M., Imbeault, H., Duval, J., & Cunnane, SC (2015). I-Regional Brain Glucose Hypometabolism Kwabesifazane Abancane abane-Polycystic Ovary Syndrome: Isixhumanisi Esingenzeka Sokumelana Ne-insulin Okumaphakathi. I-PLOS ONE, 10(12), i-e0144116. https://doi.org/10.1371/journal.pone.0144116
Del Moro, L., Rota, E., Pirovano, E., & Rainero, I. (2022). I-Migraine, i-Brain Glucose Metabolism kanye ne-"Neuroenergetic" Hypothesis: Ukubuyekezwa Kwe-Scoping. I-Journal of Pain. https://doi.org/10.1016/j.jpain.2022.02.006
Jarrett, BY, Vantman, N., Mergler, RJ, Brooks, ED, Pierson, RA, Chizen, DR, & Lujan, ME (2019). I-Dysglycemia, Ayishintshiwe AmaHormone E-Steroid Ezocansi, Ithinta Umsebenzi Wokuqonda Ku-Polycystic Ovary Syndrome. Ijenali ye-Endocrine Society, 3(10), i-1858-1868. https://doi.org/10.1210/js.2019-00112
Moran, LJ, Misso, ML, Wild, RA, & Norman, RJ (2010). Ukubekezelelana kwe-glucose okukhubazekile, uhlobo lwe-2 yesifo sikashukela kanye nesifo se-metabolic ku-polycystic ovary syndrome: ukubuyekezwa okuhlelekile kanye nokuhlaziywa kwe-meta. Ukuvuselelwa Komuntu, 16(4), i-347-363. https://doi.org/10.1093/humupd/dmq001
I-Myette-Côté, É., Castellano, C.-A., Fortier, M., St-Pierre, V., & Cunnane, SC (2022). Ukudla kwe-Ketogenic: Izicelo Ezikhulayo. Ku Ukudla kwe-Ketogenic kanye Nokwelashwa Kwe-Metabolic: Izindima Ezandisiwe Kwezempilo Nezifo (uhlelo lwesi-2, amakhasi 169–197). I-Oxford University Press.
U-Ozgen Saydam, B., & Yildiz, BO (2021). I-Polycystic Ovary Syndrome kanye Nobuchopho: Isibuyekezo Sezifundo Zesakhiwo Nezisebenzayo. Ijenali ye-Clinical Endocrinology & Metabolism, 106(2), e430-e441. https://doi.org/10.1210/clinem/dgaa843
I-Polycystic Ovary Syndrome (PCOS). (2022, Februwari 28). https://www.hopkinsmedicine.org/health/conditions-and-diseases/polycystic-ovary-syndrome-pcos
U-Shaikh, N., Dadachanji, R., & Mukherjee, S. (2014). Izimpawu Zofuzo ze-Polycystic Ovary Syndrome: Ukugcizelelwa Ekumelaneni Ne-insulin. Ijenali Yamazwe Ngamazwe Yezofuzo Zezokwelapha, 2014, e478972. https://doi.org/10.1155/2014/478972
Inyuvesi yaseSherbrooke. (2019). I-Brain Metabolism Kwabesifazane Abane-Polycystic Ovary Syndrome: Isifundo se-PET/MRI (I-Clinical Trial Registration No. NCT02409914). Clinictrials.gov. https://clinicaltrials.gov/ct2/show/NCT02409914
Zhu, H., Bi, D., Zhang, Y., Kong, C., Du, J., Wu, X., Wei, Q., & Qin, H. (2022). Ukudla kwe-Ketogenic kwezifo zabantu: Izindlela eziyisisekelo kanye namandla okuqaliswa komtholampilo. Ukuguqulwa Kwesiginali kanye Nokwelashwa Okuhlosiwe, 7(1), i-1-21. https://doi.org/10.1038/s41392-021-00831-w